Objective 1: To understand the electrophysiology behind left axis deviation (LAD)
Click to CompleteObjective 2: To be able to explain the signs one would see on an ECG with LAD
Click to CompleteObjective 3: To understand the meaning of and physiology behind left anterior fasicular block
Click to CompleteObjective 4: To be able to compare left anterior fasicular block and left posterior fasicular block
Click to CompleteTo be able to explain the pathophysiology behind right bundle branch block
Click to CompleteTo be able to distinguish the ECG changes present in RBBB and demonstrate this
understanding on example ECGs
Be able to describe the causes of RBBB
Click to CompleteTo apply the skills covered to complete the RBBB quiz
Click to CompleteIn RBBB, activation of the right ventricle is delayed as depolarisation
has to spread across the septum from the left ventricle
The left ventricle is activated normally, meaning that the early part of the
QRS complex is unchanged
The delayed right ventricular activation produces a secondary
R wave (R’) in the right precordial leads (V1-3) and a wide, slurred S wave in the
lateral leads
Delayed activation of the right ventricle also gives rise to secondary
repolarization abnormalities, with ST depression and T wave inversion
in the right precordial leads
In isolated RBBB the cardiac axis is unchanged, as left ventricular activation
proceeds normally via the left bundle branch
To be able to explain the pathophysiology behind right bundle branch block
Click to CompleteTo be able to distinguish the ECG changes present in RBBB and demonstrate this
understanding on example ECGs
Be able to describe the causes of RBBB
Click to CompleteTo apply the skills covered to complete the RBBB quiz
Click to CompleteIn RBBB, activation of the right ventricle is delayed as depolarisation
has to spread across the septum from the left ventricle
The left ventricle is activated normally, meaning that the early part of the
QRS complex is unchanged
The delayed right ventricular activation produces a secondary
R wave (R’) in the right precordial leads (V1-3) and a wide, slurred S wave in the
lateral leads
Delayed activation of the right ventricle also gives rise to secondary
repolarization abnormalities, with ST depression and T wave inversion
in the right precordial leads
In isolated RBBB the cardiac axis is unchanged, as left ventricular activation
proceeds normally via the left bundle branch
In left axis deviation (LAD) the direction of
overall electrical activity becomes distorted to the left (between -30° and -90°)
Normal Limb Leads
Left Axis deviation Limb Leads
The positive deflection in lead I to becomes more positive
and the deflection in III becomes be more negative:
- QRS is positive (dominant R wave) in leads I and aVL
- QRS is negative (dominant S wave) in leads II and aVF
- Left anterior fascicular block
- Left bundle branch block
- Left ventricular hypertrophy
- Inferior MI
- Ventricular ectopy
- Paced rhythm
- Wolff-Parkinson White syndrome
In LAFB (also known as left anterior hemiblock), impulses
are conducted to the left ventricle via the left posterior fascicle,
which inserts into the infero-septal wall of the left ventricle along its
endocardial surface
- LAD
- Small,Q waves with tall R waves in leads I and aVL
- Small R waves with deep S waves in leads II, III, and aVF
- QRS duration normal or slightly prolonged
- Prolonged R wave peak time in aVL
- Increased QRS voltage in the limb leads
LPFB is much less common than LAFB, as the broad bundle of fibres that comprise the left posterior fascicle are relatively resistant to damage when compared with the slim single tract that makes up the left anterior fascicle.
It is extremely rare to see LPFB in isolation. It usually occurs along with RBBB
in the context of a bifascicular block
Do not be tempted to diagnose LPFB until you have ruled out more significant causes of right axis deviation: i.e. acute PE, tricyclic overdose, lateral MI, right ventricular hypertrophy