To be able to explain the pathophysiology behind left
bundle branch block
To be able to distinguish the ECG changes present in LBBB
and demonstrate this understanding on example ECGs
Be able to describe the causes of LBBB
Click to CompleteTo apply the skills covered to complete the LBBB quiz
Click to Complete- To be able to explain the pathophysiology behind left bundle branch block
- To be able to distinguish the ECG changes present in LBBB
- Be able to describe the causes of LBBB
- To apply the skills covered to complete the LBBB quiz
Normally the septum is activated from left to right, producing small Q waves in the lateral leads
In LBBB, the normal direction of septal depolarisation is reversed (becomes right to left), as the impulse spreads first to the RV via the right bundle branch and then to the LV via the septum
This sequence of activation extends the QRS duration to > 120 ms and eliminates the normal septal Q waves in the lateral leads
The overall direction of depolarisation (from right to left) produces tall R waves in the lateral leads (I, V5-6) and deep S waves in the right precordial leads (V1-3), and usually leads to left axis deviation
As the ventricles are activated sequentially (right, then left) rather than simultaneously, this produces a broad or notched (‘M’-shaped) R wave in the lateral leads
Diagnostic Criteria:
- QRS duration of > 120 ms
- Dominant S wave in V1
- Broad monophasic R wave in lateral leads (I, aVL, V5-V6)
- Absence of Q waves in lateral leads (I, V5-V6; small Q waves are still allowed in aVL)
- Prolonged R wave peak time > 60ms in left precordial leads (V5-6)
The R wave in the lateral leads may be either:
‘M’-shaped
Notched
Monophasic
RS complex
In left bundle branch block, the ST segments and T waves show “appropriate discordance” — i.e. they are directed opposite to the main vector of the QRS complex
This produces ST elevation and upright T waves in leads with a negative QRS complex (dominant S wave), while producing ST depression and T wave inversion in leads with a positive QRS complex (dominant R wave)
- Aortic stenosis
- Ischaemic heart disease
- Hypertension
- Dilated cardiomyopathy
- Anterior MI
- Primary degenerative disease (fibrosis) of the conducting system (Lenegre disease)
- Hyperkalaemia
- Digoxin toxicity
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